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1. Dexmedetomidine Improves Long-term Neurological Outcomes by Promoting Oligodendrocyte Genesis and Myelination in Neonatal Rats Following Hypoxic-ischemic Brain Injury NSTL国家科技图书文献中心

Xue, Hang |  Ding, Zixuan... -  《Molecular Neurobiology》 - 2025,62(4) - 4866~4880 - 共15页

摘要:-injury, early myelination, and oligodendrocyte | Neonatal hypoxic-ischemic brain injury (HIBI |  oligodendrocyte genesis and myelination following hypoxic |  neurobehavioral outcomes. Measurements included oligodendrocyte |  survival and maturation of oligodendrocyte progenitor
关键词: Dexmedetomidine |  Oligodendrocyte |  Myelination |  Hypoxic ischemia brain injury

2. From BBB to PPP: Bioenergetic requirements and challenges for oligodendrocytes in health and disease NSTL国家科技图书文献中心

Fernandes, Milton Gu... |  Pernin, Florian... -  《Journal of neurochemistry.》 - 2025,169(1) - e16219~e16219 - 共20页

摘要: metabolites can lead to stress and oligodendrocyte injury |  oligodendrocyte injury, to support the development of novel |  in oligodendrocyte energy metabolism, as well as |  physiological properties of oligodendrocyte energy metabolism | Mature myelinating oligodendrocytes, the cells
关键词: hypoperfusion |  metabolic stress |  metabolism |  multiple sclerosis |  myelin |  oligodendrocyte injury

3. Tanshinone IIA Promotes Functional Recovery After Spinal Cord Injury by Inhibiting Neuron and Oligodendrocyte Ferroptosis Through the GPX4/ACSL4 Axis NSTL国家科技图书文献中心

Luchun,Xu |  Guozheng,Jiang... -  《Neurochemical research》 - 2025,50(3) - Article: 167~Article: 167 - 共12页

摘要:Abstract Spinal cord injury (SCI) induces |  secondary injury mechanisms. Tanshinone IIA (TIIA), a key |  severe functional impairments and involves intricate |  bioactive component of Salvia miltiorrhiza, exhibits |  neuroprotective potential, yet its role in ferroptosis
关键词: Tanshinone IIA |  Spinal cord injury |  Ferroptosis |  GPX4 |  ACSL4

4. Glycyrrhizin Attenuates White Matter Injury by Inhibiting Neuroinflammation through the HMGB1/TLR4 Pathway NSTL国家科技图书文献中心

Lou, Jia |  Ding, Bingqing... -  《Molecular Neurobiology》 - 2025,62(5) - 6070~6087 - 共18页

摘要: and OGD-induced oligodendrocyte injury through its | White matter injury (WMI) is a common |  complication of preterm birth, potentially resulting in long | -term behavioral and motor abnormalities. The |  objective of this study is to investigate the
关键词: White matter injury (WMI) |  Oligodendrocyte |  Glycyrrhizin (GLY) |  Hypoxia |  Inflammation

5. Neuroinflammation: An Oligodendrocentric View NSTL国家科技图书文献中心

Festa, Lindsay K. |  Jordan-Sciutto, Kell...... -  《Glia》 - 2025,73(6) - 1113~1129 - 共17页

摘要: accumulating evidence that injury to the oligodendrocyte |  viral infections, converges on oligodendrocyte injury |  perinatal white matter injury, traumatic brain injury, and |  impact of inflammation on the oligodendrocyte lineage | Chronic neuroinflammation, driven by central
关键词: human immunodeficiency virus |  multiple sclerosis |  oligodendrocytes |  perinatal white matter injury |  SARS-CoV2 |  traumatic brain injury |  SARS‐CoV2

6. Unraveling the role of oligodendrocytes and myelin in pain NSTL国家科技图书文献中心

Kim, Woojin |  Angulo, Maria Cecili... -  《Journal of neurochemistry.》 - 2025,169(1) - e16206~e16206 - 共14页

摘要: pain following peripheral nerve injury, spinal cord |  injury (SCI), chemotherapy, and HIV infection. Studies |  response to injury can induce or exacerbate pain. An |  increase in endogenous oligodendrocyte precursor cells | , oligodendrocyte apoptosis in brain regions such as the medial
关键词: central nervous system |  myelin |  oligodendrocyte precursor cells |  oligodendrocytes |  pain

7. Myelination potential and injury susceptibility of grey versus white matter human oligodendrocytes NSTL国家科技图书文献中心

Qiao-Ling,Cui |  Abdulshakour,Mohamma...... -  《Brain》 - 2025,148(3) - 921~932 - 共12页

摘要: oligodendrocyte lineage cells both during development and in |  susceptible than white matter A2B5-positive cells to injury |  oligodendrocytes were lower expression of mature oligodendrocyte |  oligodendrocyte lineage genes. Genes and pathways with increased |  genes shared between the stress/injury and myelination
关键词: ensheathment |  grey matter |  white matter |  molecular signatures |  oligodendrocyte lineage

8. Oligodendrocyte differentiation on murine decellularized brain tissue NSTL国家科技图书文献中心

Nishimura, Hinata |  Kerever, Aurelien... -  《Neuroscience Letters》 - 2025,846 - 138079~138079 - 共8页

摘要: oligodendrocyte precursor cells (OPCs). The GPR17-expressing |  oligodendrocyte development, acts as a reservoir responding to |  brain injury and ischemia. GPR17 plays a complex role |  in oligodendrocyte maturation and response to |  injury; its activation promotes differentiation into
关键词: Oligodendrocyte |  Decellularization |  Extracellular matrix |  Chondroitin sulfate chains

9. Dispensable regulation of brain development and myelination by the immune‐related protein Serpina3n NSTL国家科技图书文献中心

Meina Zhu |  Yan Wang... -  《Journal of neurochemistry.》 - 2025,169(2) - e16250~e16250 - 共17页

摘要: appear to interfere with oligodendrocyte |  alleviates oxidative injury and diminishes cell senescence |  and regulates oligodendrocyte health under injured |  functional significance in the CNS under disease/injury | Abstract Serine protease inhibitor clade A
关键词: brain development |  differentiation |  myelination |  oligodendrocytes |  oxidative stress |  Serpina3n

10. A new player in the game: identification of C1ql1 as a novel factor driving OPC differentiation NSTL国家科技图书文献中心

Van Broeckhoven, Jan... |  Mussen, Femke... -  《The FEBS journal》 - 2025,292(1) - 47~51 - 共5页

摘要:). Following injury, these cells are prone to death, leading |  neurodegeneration. Upon injury, the damaged CNS repopulates the |  lesion with oligodendrocyte precursor cells (OPCs) that | Oligodendrocytes (OLGs) are the myelin | -producing cells in the central nervous system (CNS
关键词: complement component 1, q subcomponent-like-1 |  differentiation |  myelination |  oligodendrocyte precursor cells |  complement component 1, q subcomponent‐like‐1
检索条件oligodendrocyte injury
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